Please use this identifier to cite or link to this item: http://nopr.niscair.res.in/handle/123456789/5040
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dc.contributor.authorMailankot, Maneesh-
dc.contributor.authorJayalekshmi, H-
dc.contributor.authorChakrabarti, Amit-
dc.contributor.authorAlang, Neha-
dc.contributor.authorVasudevan, D M-
dc.date.accessioned2009-07-01T03:56:31Z-
dc.date.available2009-07-01T03:56:31Z-
dc.date.issued2009-07-
dc.identifier.issn0975-1009 (Online); 0019-5189 (Print)-
dc.identifier.urihttp://hdl.handle.net/123456789/5040-
dc.description608-610en_US
dc.description.abstractEthanol intoxication resulted in high extent of lipid peroxidation, and reduction in antioxidant defenses (decreased GSH, GSH/GSSG ratio, and catalase, SOD and GPx activities) and (Na+/K+)-ATPase activity in kidney. Alpha-tocopherol treatment effectively protected kidney from ethanol induced oxidative challenge and improved renal (Na+/K+)-ATPase activity. Ethanol induced oxidative stress in the kidney and decreased (Na+/K+)-ATPase activity could be reversed by treatment with ascorbic acid.en_US
dc.language.isoen_USen_US
dc.publisherCSIRen_US
dc.sourceIJEB Vol.47(07) [July 2009]en_US
dc.subjectAlpha-tocopherolen_US
dc.subjectEthanolen_US
dc.subjectKidneyen_US
dc.subjectOxidative stressen_US
dc.subjectNa+ / K+-ATPaseen_US
dc.titleEffect of alpha-tocopherol supplementation on renal oxidative stress and Na+/K+-adenosine triphosphatase in ethanol treated Wistar ratsen_US
dc.typeArticleen_US
Appears in Collections:IJEB Vol.47(07) [July 2009]

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